Authors | Mohsen Mohammadnia Ahmadi,Mohsen Nematy,Mohammad Ali Sardar |
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Journal | Obesity Medicine |
Page number | 1-7 |
Serial number | 17 |
Volume number | 17 |
Paper Type | Full Paper |
Published At | 2020 |
Journal Grade | ISI |
Journal Type | Electronic |
Journal Country | Belgium |
Journal Index | Scopus |
Abstract
Background: Thepurposeofthisstudywastodeterminetheeffectofhigh-intensityintervaltrainingwithand withoutOligopinsupplementationonsomeinflammatoryindicesandliverenzymesinobesemaleWistarrats withnon-alcoholicfattyliverdisease.Methods:36adultmaleWistarratsweredividedintofivegroupsincluding normal diet sedentary (ND-SED), high-fat diet sedentary (HFD-SED), high-fat diet & high-intensity interval training(HFD-HIIT),high-fatdiet&Oligopin(HFD-O),high-fatdiet&high-intensityintervaltraining&Oligopin (HFD-HIIT+O).Firstly,inordertoinducefattyliver,high-fatfoodwasusedfor12weeks. Results: EightweeksofHIITledtoasignificantdecreaseofinterlucin-6(IL-6),tumornecrosisfactor-alpha(TNFα)andaspartateaminotransferase(AST)inplasmalevelsandasignificantincreaseofVO2max.Besides,eightweek administration of supplementary Oligopin resulted in a significant decrease of AST and alanine transaminase(ALT)whilenosignificantchangewasseeninthelevelsofTNF-αandIL-6.Inaddition,HIITwithan administrationofOligopinsupplementationledtoagreaterdecreaseinAST,ALT,IL-6,TNF-α. Conclusions: This study suggests that HIIT and Oligopin result in improving NAFLD disease caused by HFD. However,HIITandOligopinsupplementationarethemosteffectiveifcombined.
tags: High-intensity interval training, Oligopin, Inflammatory indices, Non-alcoholic fatty liver disease